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OMIA:001354-9925 : Muscular hypertrophy (double muscling), Callipyge in Capra hircus (goat)

In other species: sheep

Categories: Muscle phene

Links to possible relevant human trait(s) and/or gene(s) in OMIM: 176290 (gene) , 613648 (gene)

Single-gene trait/disorder: unknown

Disease-related: unknown

Cross-species summary: Abnormal increase in muscular tissue caused entirely by enlargement of existing cells (in contrast to muscular hyperplasia, in which the abnormal increase in muscular tissue is due to the formation and growth of new, normal muscle cells)

Genetic engineering: Yes - variants have been created artificially, e.g. by genetic engineering or gene editing
Have human generated variants been created, e.g. through genetic engineering and gene editing

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Cite this entry

Nicholas, F. W., Tammen, I., & Sydney Informatics Hub. (2025). OMIA:001354-9925: Online Mendelian Inheritance in Animals (OMIA) [dataset]. https://omia.org/. https://doi.org/10.25910/2AMR-PV70

Reference

2025 Hajian, M., Pirali, A., Moghaddam, S.H.H., Moradi-Hajidavaloo, R., Varnosfaderani, S.R., Jozi, M., Izadi, T., Tanhaie-Vash, N., Kues, W., Nasr-Esfahani, M.H., Jafarpour, F., Eghbalsaied, S. :
Efficient gene editing of BMP15, GDF9, and MSTN-but not the imprinted CLPG gene-in goat embryos via electrotransfection and handmade cloning. Funct Integr Genomics 25:150, 2025. Pubmed reference: 40637794. DOI: 10.1007/s10142-025-01644-8.

Edit History


  • Created by Imke Tammen2 on 12 Jul 2025